Some medications are known to produce acne. Some cortisones, few anti-tuberculosis medications and some anti-epileptic and anti-seizure medications can produce acne. Also medications that include anabolic steroids, and lithium and iodine-based medications.

Hormone preparations like contraceptive solutions and older oral contraceptives can make acne worse. Other medications known to boost acne include certain antidepressants, and cyclosporin.

Thyroid Medications: recommended to activate the thyroid gland in patients with low thyroid function. Acne is a side effect.

Disulfuram - recommended for alcoholic patients trying to achieve sobriety. Regular use can produce acne.

Immunosuppressants - recommended to suppress the immune system; primarily used to prevent organ rejection in patients awaiting transplants. Immune suppression permits bacteria to flourish, including the bacteria that causes acne, P. acnes.

Oral Vitamin A: Retinoids (derivatives of vitamin A) are used topically and orally to alleviate acne under medical supervision. Vitamin A does not alleviate acne. If you take excessive vitamin A, hoping that it will cure acne, your health may become worse. Keep in mind that Vitamin A in excess quantity can have adverse effects on the body.

Hereditary: Acne can be hereditary. If your parents had acne, you may be more prone to it.

Hormonal Variations: Hormonal changes produce acne. The hormone androgen is responsible for excess secretion of sebum. Women can have acne episodes during menstruation and pregnancy.

Acne-Like Conditions: Some other ailments like folliculitis may appear like acne. There are many other conditions that may look like acne. Some of them are perioral dermatitis, rosacea, keratosis pilaris, etc. Always visit a dermatologist instead of trying self-treatment.

Usual concerns about treating acne

Excessive sebum secretion: At puberty, increasing levels of androgens, the major sebotrophic hormones, begin to drive an increase in sebum secretion. However, while androgenic stimulation is important in the pathogenesis of acne, the average acne patient does not have significant endocrine irregularities. Hormonal therapy is not recommended in the initial treatment of mild to moderate acne, although women who use oral contraception may be candidates for anti-androgen therapy early in the course of treatment.

Aberrant desquamation of the follicular epithelium: In acne, keratinocytes hyperproliferate and accumulate within the sebaceous follicle. As these aberrantly desquamated cells accumulate in the sebaceous follicle, they produce microcomedo development. The microcomedo is the precursor to all acne lesions and is present in 80% of acne papules but is invisible to the unaided eye. However, as the already clogged follicle begins to fill with lipids, microbes and cell fragments, the microcomedo changes to open or closed comedones (blackheads and whiteheads, respectively), both of which are non-inflammatory lesions. If P. acnes grows, inflammatory promoters are produced and inflammatory papules and pustules occur.

Bacterial growth: The microenvironment of the follicle in acne is conducive to colonization with P. acnes. This leads to inflammation and the production of the noticeable papules and pustules with which acne patients commonly present to dermatologists.

Inflammation: Inflammation in acne happens as a result of humoral and cellular immune reactions to P. acnes growth.

The best solution to treat acne scars and other skin blemishes comes now in the form of a new skin care product, elaborated with the best natural components to rejuvenate your skin.

- Danna Finnerand

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